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Old 07-26-2018, 12:34 PM
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I know this has pros & cons but advocates for grain-free diets (as much as poss) seem to agree that, tho the brain uses alot of carbs, that gluconeogenesis can fulfill these needs. Anyone know their biochem?
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Old 07-26-2018, 03:04 PM
Location: Sector 001
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The body will eventually adapt to a zero carb diet and aerobic endurance can be quite good if not superior, but you have to ask what the usefulness of such a diet is for you. Moderate amounts of carbs are not harmful, say up to 100 grams per day if you don't exercise, and more depending on how much cardio you do. Pure glucose (which is what starch ends up as) can be used by any cell in the body as opposed to fructose (50% of sugar) which must be processed by the liver. Best time to eat starches is before cardiovascular exercise because you'll burn a lot of them off before the body can even store them as fat.

We might be omnivores but yes most people eat too many carbohydrates and it's carbohydrates in excess that cause most health problems, especially if you combine carbohydrates with fats and overeat calories. Fat builds up in the liver and pancreas and leads to metabolic syndrome. This is especially so if you don't consume choline (found in eggs) or betane (found in whole wheat).. in that regard if you are going to eat carbs, whole wheat is probably the healthiest, and not rice, oats, or a ton of refined stuff. Or just take a choline bitartate supplement or eat eggs. Problem solved.


You only need to look at what happens to dogs, cats, horses, and other animals when you feed them cheap, high carb foods.. health tends to suffer. Even livestock which are grass eaters suffer negative effects from low fiber refined carbs. People want to put the blinders on and pretends it's the fat that's the problem while they keep shoveling down the triscuits and eating their hot pockets and pizza. Portion size is very important when it comes to prediabetes or type 2 diabetes.. a whole dairy queen large blizzard is not really good for anyone but some people can genetically handle the sugar load better than others. A type 2 would likely spike above 300-400 with a large blizzard and such items are not advised.

Low carb reduces blood sugar and can "treat" type 2 diabetes but is not really a cure.. without removing the fat from the liver and pancreas you will not have any sort of cure. Lose weight, eat a lower carb diet, and get plenty of choline and betane while you are in a calorie deficit so your body targets the liver and organs when removing fat. Get a blood glucose tester (beware most of them on the market read 10-20 points higher than your actual, lab tested value).. the only tester I found to be really accurate is discontinued.. it was a "trueresult" meter.

Last edited by stockwiz; 07-26-2018 at 03:39 PM..
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Old 07-26-2018, 03:49 PM
Location: McAllen, TX
2,900 posts, read 1,928,267 times
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^^ Even though the facts are there in black and white, you will get some people denying this happens.

This is the actual study
Effects of Step-Wise Increases in Dietary Carbohydrate on Circulating Saturated Fatty Acids and Palmitoleic Acid in Adults with Metabolic Syndrome

No matter what happens, many will hold on to the idea that saturated fats are bad. There IS NO direct correlation between dietary saturated fat and heart disease. The title of the article that stockwiz posted a link to is "Adding Carbs to the Diet Increases Saturated Fat in Blood"

Most will not click on the link, here is an excerpt.

Recent meta-analyses have found no association between heart disease and dietary saturated fat; however, higher proportions of plasma saturated fatty acids (SFA) predict greater risk for developing type-2 diabetes and heart disease. These observations suggest a disconnect between dietary saturated fat and plasma SFA, but few controlled feeding studies have specifically examined how varying saturated fat intake across a broad range affects circulating SFA levels. Sixteen adults with metabolic syndrome (age 44.99.9 yr, BMI 37.96.3 kg/m2) were fed six 3-wk diets that progressively increased carbohydrate (from 47 to 346 g/day) with concomitant decreases in total and saturated fat. Despite a distinct increase in saturated fat intake from baseline to the low-carbohydrate diet (46 to 84 g/day), and then a gradual decrease in saturated fat to 32 g/day at the highest carbohydrate phase, there were no significant changes in the proportion of total SFA in any plasma lipid fractions. Whereas plasma saturated fat remained relatively stable, the proportion of palmitoleic acid in plasma triglyceride and cholesteryl ester was significantly and uniformly reduced as carbohydrate intake decreased, and then gradually increased as dietary carbohydrate was re-introduced. The results show that dietary and plasma saturated fat are not related, and that increasing dietary carbohydrate across a range of intakes promotes incremental increases in plasma palmitoleic acid, a biomarker consistently associated with adverse health outcomes.
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